WITH NO MEDICAL CAUSES
TIME:1999, MARCH before noon
I was preaching to the English speaking group with Blessed Sacrament Exposed… after the hemorrhage.. I was unconscious and many other fatal complications, but I preached again for another 45 minutes to complete the session …
Scanned at Palanpur, Later with medical check up in Ahmedabad took a flight to Kochi diagnosed SAH with the scanning and then was diagnosed perfectly normal after 4 days during Angiography. Thanks to al who prayed for me for this miraculous cure…..
PLACE: ABU ROAD, RAJASTHAN DURING PREACHING MINISTRY AT AHMEDABAD, SIROHI
EXPERIENCES AT LISIEUX HOSPITAL AT KOCHI
The immediate danger due to subarachnoid hemorrhage, whether traumatic or spontaneous, is ischemia. Ischemia refers to tissue damage caused by restricted or blocked blood flow. The areas of the brain that do not receive adequate blood and oxygen can suffer irreparable injury, leading to permanent brain damage or death. An individual who survives the initial hemorrhage is susceptible to a number of complications in the following hours, days, and weeks.
The most common complications are intracranial hypertension, vasospasm, and hydrocephalus. Intracranial hypertension, or high pressure within the brain, can lead to further bleeding from damaged blood vessels; a complication associated with a 70% fatality rate. Vasospasm, or blood vessel constriction, is a principal cause of secondary ischemia. The blood vessels in the brain constrict in reaction to chemicals released by blood breaking down within the subarachnoid space. As the blood vessels become narrower, blood flow in the brain becomes increasingly restricted. Approximately one third of spontaneous subarachnoid hemorrhages and 30-60% of traumatic bleeds are followed by vasospasm. Hydrocephalus, an accumulation of fluid in the chambers of the brain (ventricles) due to restricted circulation of cerebrospinal fluid, follows approximately 15% of subarachnoid hemorrhages. Because cerebrospinal fluid cannot drain properly, pressure accumulates on the brain, possibly prompting further ischemic complications.